Preliminary experiments indicate conolidine may perhaps inhibit particular ion channels, minimizing neuronal excitability and restricting ache signals. This mechanism is especially applicable in neuropathic ache, where by irregular signaling causes persistent distress. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to suffering notion. Computation... https://robertn517hzq3.buscawiki.com/user